Papermill Alarm

FREEMIUM
By Clear Skies | Updated 3ヶ月前 | Science
Popularity

7.5 / 10

Latency

3,623ms

Service Level

100%

Health Check

N/A

README

Using the API

The API runs some resource-intensive deep-learning methods. Resources are scaled up automatically as request rates rise. However, there is a delay between request-rates rising and new resources being created which can trigger 504 errors. Such errors are rare, but if you receive a 504 error, please wait 60s before resuming use of the API.

The API takes a very simple input via a POST request. You may request up to 4 articles at a time. E.g.



import requests

url = "https://papermill-alarm.p.rapidapi.com/"

payload = {
			"id": "retracted_article_id2",
			"title": "Silencing circANKRD36 protects H9c2 cells against lipopolysaccharide-induced injury via up-regulating miR-138",
			"abstract": "Background: Myocarditis refers to the inflammatory process that affects the muscle tissue of the heart. Persistent myocardial inflammation promotes myocardial cell damage, which ultimately leads to heart failure or death. Therefore, we aimed to explore the functional impacts of circANKRD36 on myocarditis. Methods: H9c2 cells were pre-treated with lipopolysaccharide (LPS). Viability and apoptosis were evaluated utilizing CCK-8 assay and flow cytometry. Inflammatory cytokines mRNA expression and production were assessed by qRT-PCR and ELISA. Western blot was utilized to distinguish apoptosis and inflammatory related factors expression. Sequentially, the above mentioned parameters were reassessed when overexpressed miR-138. Results: LPS declined viability and as well as raised apoptosis and inflammatory injury in H9c2 cells. Silencing circular RNA ANKRD36 (si-circANKRD36) alleviated apoptosis and inflammatory injury induced by LPS. MiR-138 expression was suppressed by LPS and elevated by si-circANKRD36. Increase of viability and inflammatory injury induced by si-circANKRD36 was alleviated by down-regulation of miR-138. Eventually, si-circANKRD36 inhibited p38MAPK/NF-κB pathway which activated by LPS via up-regulating miR-138. Conclusion: Si-circANKRD36 exerted its anti-apoptosis and anti-inflammatory function under the condition of LPS in H9c2 cells through p38MAPK/NF-κB pathway and up-regulation of miR-138."
		}

headers = {
	"X-RapidAPI-Key": os.getenv("X-RAPIDAPI-KEY"),
	"X-RapidAPI-Host": "papermill-alarm.p.rapidapi.com",
	"Content-Type": "application/json"
}

response = requests.request("POST", url, json=payload, headers=headers)

print(response.text)
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Rapid account: Clear Skies
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